rumitoid wrote:
I am highly prejudice on this issue. Both my mother and sister died in agony for an extended period of time from cancer. If either had asked for help to the other side, I would have helped, though never suggest it. (As I know now, a peaceful extra dose of morphine would have done it.) But we were very Catholic. It was thought to be murder. And there was also the belief that God would find it in his mercy for a cure. I guess more than it would be murder as a deterrent, turning one's back on hope and God's power was a more effective argument.
In 1989, I went for a Hospice Degree. What I learned over the next four years, both in class and in hospitals, made me advocate for assisted suicide, even in cases of Alzheimer. But I am still not totally comfortable with my belief. Mostly that it can become a slippery slope and, like the presence of a******n, might inculcate a decrease in the sacredness of life. Or not. Maybe increase it.
If you were in extreme agony and told there was no possible cure, your family forced to see you tortured day after day after day and unable to help, what would you do and why?
I am highly prejudice on this issue. Both my mothe... (
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Curious, how the Health Department is both morally and financial bankrupt. They would prefer to balance their budget over a mountain of corpses than find a cure for "chronic" diseases. What ever happened to the Sanctity of life? When it comes to the government, it is cheaper to k**l us than cure us.
Fortunately, there is new private pharmaceutical research in strengthening immunological responses by enhancing human bone marrow's creation of antibodies. Check out the TREM-2 pharmaceuticals:https://ghr.nlm.nih.gov/gene/TREM2TREM2 gene triggering receptor expressed on myeloid cells 2
Normal Function:
The TREM2 gene provides instructions for making a protein called triggering receptor expressed on myeloid cells 2. As its name suggests, this protein is made in myeloid cells, which are cells produced in bone marrow. The TREM2 protein is found on the cell surface, where it interacts with the protein produced from the TYROBP gene. The TREM2 and TYROBP proteins form a complex that t***smits chemical signals to activate the cell.
The TYROBP-TREM2 complex was first identified in the i****e s****m. This complex is involved in the growth and development of several types of immune cells, particularly dendritic cells. The TYROBP-TREM2 complex activates these cells, triggering an inflammatory response to injury or disease.
The TYROBP-TREM2 complex also activates cells in the skeletal system and in the brain and spinal cord (central nervous system). In the skeletal system, the complex is found in osteoclasts, which are specialized cells that break down and remove (resorb) bone tissue that is no longer needed. These cells are involved in bone remodeling, which is a normal process that replaces old bone tissue with new bone. In the central nervous system, the complex appears to play an important role in immune cells called microglia. These cells protect the brain and spinal cord from foreign invaders and remove dead nerve cells and other debris. Although the TYROBP-TREM2 complex plays a critical role in osteoclasts and microglia, its exact function in these cells is unclear.
TREM 2 definition: Triggering Receptor Expressed on Myeloid cells
A candidate regulatory variant at the TREM gene cluster associates ...
https://www.pubfacts.com/.../A-candidate-regulatory-variant-at-the-TREM-gene-cluster-...
Results: A variant within a DNase hypersensitive site 5' of TREM2, ... young men after six weeks supplementation of whey protein isolate and resistance training.
US20160251434A1 - Novel receptor trem (triggering receptor ...
https://patents.google.com/patent/US20160251434TREM-2 is also a t***smembrane glycoprotein expressed selectively on mast cells ... and mammary gland-specific promoters (e.g., milk whey promoter; U.S. Pat.
US20130028901A1 - Novel receptor trem (triggering receptor ...
https://patents.google.com/patent/US20130028901https://patents.google.com/patent/US20130028901TREM-2 is also a t***smembrane glycoprotein expressed selectively on mast cells ... and mammary gland-specific promoters (e.g., milk whey promoter; U.S. Pat.
Novel receptor trem (triggering receptor expressed on myeloid cells) and uses thereof
Abstract
Novel activating receptors of the Ig super-family expressed on human myeloid cells, called TREM(s) (triggering receptor expressed on myeloid cells) are provided. Specifically, two (2) members of TREMs, TREM-1 and TREM-2 are disclosed. TREM-1 is a t***smembrane glycoprotein expressed selectively on blood neutrophils and a subset of monocytes but not on lymphocytes and other cell types and is upregulated by bacterial and fungal products. Use of TREM-1 in treatment and diagnosis of various inflammatory diseases is also provided. TREM-2 is also a t***smembrane glycoprotein expressed selectively on mast cells and peripheral dendritic cells (DCs) but not on granulocytes or monocytes. DC stimulation via TREM-2 leads to DC maturation and resistance to apoptosis, and induces strong upregulation of CCR7 and subsequent chemotaxis toward macrophage inflammatory protein 3-β.
TREM-2 has utility in modulating host immune responses in various immune disorders, including autoimmune diseases and allergic disorders.This would include HIV infections. Lordy, Lordy, wake up all ye liberal dingbats. The government does not love you!